Once again I came across some claims without evidence whether or not the Neanderthals were rh positive or rh negative.
Having grown tired of senseless discussions, I have decided to contact the Max Planck Institute in Germany which has conducted the Neanderthal Genome Project.
Svante Pääbo is the head of the department and I have contacted him.
Upon contacting him, I received the following email from one of his assistants:
Svante forwarded me your request. The Denisova and Altai Neandertal are
homozygous for the ancestral “A” variant at position 25629943 on
chromosome 1 that determines rhesus type in modern humans. This variant
means that both are likely rhesus positive.
One thing I need to first stress: Don´t focus on countries as you can encounter a lot of variations from region to region. A great example is Chile where in some area the percentage of rh negatives tops 20 percent and in other cannot even reach 1 percent.
What I am interested in is getting regions noticed which may help us by pointing us into the right direction to find more answers on our origin.
Here are some regions and countries and other places which simply stand out as the number of rh negatives is simply topping either the frequencies in the world or the percentages prevalant amongst surrounding neighbors:
1) Basque Country
Basque Country is the obvious choice.
But looking at both, the frequency of the rh negative blood factor and the presence of the gene, regional variations can be strong. One thing to notice is that in the Spanish part of Basque country the frequency is higher than in the French part indicating a possibility of more marriages being formed between Basques and French than Basques and Spanish.
2) Bunshouten & Spakenburg, Netherlands
With less than 20,000 people making up their population, this is not a huge region. But nevertheless, most than 50% there carry the rh negative gene.
There are probably other regions somewhere not yet discovered, but this one stands out as of now.
3) Rhone Valley, Switzerland
Safien, Tenna and Versam especially.
Another early reference is the legend of the Curse of Tenna. In 1769 a judge in the small Swiss village of Tenna condemned an innocent man to death. The legend states that this act led to the inflicting of a curse upon the judge and his family. This curse was believed by the citizens of Tenna to be the cause of a serious bleeding disorder, sometimes leading to bleeding to death, which afflicted the family for generations. This was, in fact, a family with hereditary hemophilia, the oldest and largest such family ever described comprising 3072 members, 55 who had hemophilia. It turns out that the family disease is hemophilia B rather than the more common hemophilia A.
You may ask yourself why I would pick a country with around 7% rh negatives.
a) 7% is huge for Africa.
b) The origin of the Ethiopians is very interesting. A high frequency of haplogroup J connects them to both, the Sumerians and the Basques.
c) Reading what Roman historian Tacitus wrote about the ancient Aethiopians brings up the question who their ancestors were and what they looked like. According to him, they were “very light”. Could it be that a tribe of red haired, blue eyed and pale skinned Sumerians migrated into that region and that 1,000s of years of mixing with neighbors is responsible for their unique look?
Here is a map of how the world looked according to Greek historian Herodotus who lived from 484–425 BC:
Back then there were less man made country making it a lot easier to look for traces of migration.
On national levels these countries except Chile are at around 19% rh negatives which is huge. But once again, you need to look at the ethnic groups responsible. Australia has a lot of roots in Ireland and Northern Ireland. In Spain, the Basques are mainly responsible for the high frequencey. In Brazil, it´s likely that we have the same Basque heritage present even though one must consider that the data we have been looking at could not represent the whole nation as the ethnic variety in Brazil also calls on a need to examine all regions more closely.
Chile has a huge Basque population and it is reported that amongst the higher income class the amount of people with blood type A and the amount rh negatives is far larger than that of the European average.
But with their indigenous population having in some cases less than 1% rh negatives, their national numbers are nowhere close to the Australian, Spanish and possibly Brazilian averages.
Now what about the Berbers and the Atlas region of Morocco?
Truth is, I have yet to see evidence other than the claim by Dr. Luigi Luca Cavalli-Sforza.
Truth is that I have also grown to question some of his work, especially the claim that the world is moving towards a 100% rh positive population.
Could it be that he was right about the Berbers?
Of course. But looking at nations with high percentages of Berbers, the national averages of rh negatives do not indicate any significant amounts. On the contrary, some of the numbers are on the lower end.
Our research continues. A lot of fabrications left to ignore and a lot of needles in the haystacks to accidentally step on.
What is our origin?
It could be Sumer. Or Mesopotamia. Or Babylon.
The most important thing is to disconnect from anything that is man made and simply follow the path of DNA and blood.
And the closer we get to whatever the truth is, the easier it becomes as those articles making false claims become a lot easier to look through and areas of interest always seem to spark a new flame in those who actively research and don´t have any other interest than finding out what is, what was and what could be in the future compared to those who simply absorb “information” because it´s the first thing they find on Google or because someone who is titled to know these things makes a claim that they themselves do not dare question.
Yes, we have all heard about the blood type diet. But what types of foods are best for rh negatives?
Note: This is only the beginning. You can enter the forum yourself and see the views. Also note that some subjects are in the private section of the forum, so while they may reach a high number of readers interest wise, they are only accessible to members of the forum.
This is one of the reasons why blood type dating makes the most sense:
Children conceived through sperm donation have a birth defect rate of almost a fifth compared with the general population. This may be explained by the fact that sperm banks only accept donors who have good semen quality, and because of the rigorous screening procedures which they adopt, including a typical age limitation on sperm donors, often limiting sperm donors to the ages of 21-39 (see paternal age effect), and genetic and health screening of donors. In addition, sperm banks may try to ensure that the sperm used in a particular recipient woman comes from a donor whose blood group and genetic profile is compatible with those of the woman.
Sperm donation is also used in cases of rhesus incompatibility. This particularly occurs where a woman has a blood type which is rhesus negative, and where her partner is rhesus positive. The woman’s body may reject a fetus if it has rhesus positive blood. Anti D injections have been developed and may be used to attempt to avoid this, and these are usually automatically given to rhesus negative women immediately after they give birth to their first child. However, in the past this was either not possible or was not always routinely undertaken where a woman gave birth or had an abortion and she may have trouble carrying a child later in life. Furthermore, for some women, the anti D injection does not provide the entire solution, particularly where there is a medical history of complications during pregnancy which risk the woman’s blood and that of the fetus becoming mixed. In such cases, sperm from a rhesus negative donor can provide the solution and a woman may be able to conceive and carry a pregnancy to full term when otherwise this would not be possible. For this reason, sperm from rhesus negative sperm donors is often in great demand, particularly those with the O negative blood group who are universal donors.
There is so much misinformation out there and since it is mandatory for everyone to make an educated decision, I have decided to make the health, pregnancy and science related sections of my forum public so everyone is able to access the information pertaining to rh negative pregnancies without signing up and without logging in.
You can access the information by visiting our forum here.
Rhesus negative pregnancies have been at the forefront of our discussion for years, so let me go ahead and highlight some information which (unfortunately) is not common knowledge (yet) in order to clear up confusion and answer a few questions you might possibly have:
1) Every pregnancy is different and whether or not you need the shot depends on many different factors:
a) Did your antibodies build?
Truth is: You can, as an rh negative, be transfused once with rh positive blood as long as the ABO is compatible. Your antibodies will build because of it, so you will need an Anti-D shot before having contact with rh positive blood again.
This means: If you have had your antibodies built already, because at one point you have been in touch with rh positive blood, you do not need your Anti-D shot a few weeks into your first pregnancy.
You need it before the pregnancy.
If you do not get it, a “chemical pregnancy” can likely occur if the fetus is rh positive.
In a chemical pregnancy, your pregnancy test shows up positive and later you go to the doctor and he tells you that there is no fetus.
IF you believe to have had contact with rh positive blood previously, even though your medical records may not be available and/or IF you have had chemical pregnancies before, please demand that you get an antibody screening and if you have the intention to be pregnant again, then get your Anti-D shot NOW!
b) Are you and your partner both true Rh D-negatives?
“Rh negative” is the “common term”. D-negative is the term used by the scientific community pinpointing the lack of the D antigen.
Note: A Weak-D or a Partial-D can both show up as rh negatives or rh positives in blood tests. If both parents are true D-negatives there is zero possibility for a child to turn out anything else but D-negative.
If both you and your partner are true D-negatives, you do not need the shot and you will not need the shot after the pregnancy.
2) If your antibodies didn’t build before the pregnancy, you will not need the shot until after the pregnancy.
In Europe, the shot is being administered after the pregnancy which in the U.S. a few weeks into the pregnancy.
If this is your first pregnancy, you will not need the shot, but again: An antibody screening is necessary to ensure that you have not yet had your antibodies built for whatever reason.
Only if the baby is rh positive will you need the Anti-D shot after the pregnancy.
If the baby is Rh D-negative like you, you will not need it.
Here is an ongoing discussion for you to read up more on when and if you, as an Rh D-negative woman who is or wants to become pregnant, needs the shot:
Please note: The medical industry is not always working for individual’s best interests, but rather operating by standard operating procedures which can provide you treatment you do not need and which can possibly carry side-effects while other times not giving you the treatment that you need.
We are not physicians and only as accurate as the information provided to us or found by us, so we also need you to send us information that corrects mistakes or that adds necessary information to our research.
If you believe to have information that can benefit us, please do not hesitate to bring it to our attention.
Our forum has a 100 percent no trolling policy, so we only allow people to register and post who have been previously screened.
If you are interested in becoming a member, please do not hesitate to register here.
After you register, send us a quick message and we will grant you full access.
The best way to do that is to inbox us via our public Facebook page here.
In the past few years, the topic of whether or not the Anti D shot necessary for rh negative mothers of rh positive children may be a contributing factor to the autism epidemic has been coming up again and again.
Pharmaceutical industries, several doctors and scientists claim there is no proof that vaccines and autism can be linked while other doctors and scientists claim that there are indicators that they could be linked.
I am wondering if any of you has studies at hand that can contribute to our ongoing discussion to help us determine whether or not ingredients, former and/or current could be responsible for an ASD jump from 1 in 5,000 to 1 in 88 within a few decades.
Please only post scientific material with credible studies.
One of the things I have been doing lately is look for quotes by rh negative celebrities, but truth is, a lot of people who I know personally and are rh negative, have quotes of their own just as good if not better.
If you are new to this blog and need some inspiration or have something to share, please join us on Facebook and inspire and be inspired.
The following paragraph explains it in such easy to understand terms, I am posting it in here for reference since this is a question very commonly asked:
There are several blood groups including A, B, AB, O and some others which are more rare. Blood is also either rhesus positive or rhesus negative. The rhesus type you have is dependant on the blood group of your parents. When a woman who has rhesus negative blood carries a rhesus positive pregnancy in her uterus, the blood cells from her pregnancy can enter her blood stream. Her immune system will treat the blood from the foetus as foreign and will respond by making antibodies, which will destroy the blood cells of the foetus.
By giving an injection of rhesus antibodies known as Anti-D, the woman’s immune system is prevented from making antibodies against future pregnancies. Only women who are a rhesus negative group will receive an Anti-D injection, this is given straight after an abortion or after a full term pregnancy.
Source: What does it mean if I am Rhesus Negative and what is Anti-D?
4) Neanderthals had a larger cranial capacity than humans today which would match reports that rh negs are on the average more intelligent than rh positives.
5) Neanderthal remains have been found in the areas where the percentages of rh negatives seem to be highest, with the only exception of Morocco and other areas where the Berbers live.
“It is likely that Neanderthals were absorbed by modern humans,” he said. “My research suggests that they were a different kind of human, but humans nonetheless. We are more brothers than distant cousins.”
WASHINGTON — We have met Neanderthal and he is us – at least a little. The most detailed look yet at the Neanderthal genome helps answer one of the most debated questions in anthropology: Did Neanderthals and modern humans mate?
Early humans and Neanderthals (Homo neanderthalenis) co-existed, and researchers have long searched for evidence that the two groups mated.
Labuda got his first sign of the interbreeding about a decade ago when he discovered a snippet of DNA on the X chromosome found only in non-Africans and whose origin was unknown. (X chromosomes are sex chromosomes; women have two and men have one, paired with a Y chromosome.)
But until 2010 the group didn’t have anything to compare the snippet with. That year, the Neanderthal genome was sequenced, and a team of researchers (not including Labuda) reported in the journal Science that between 1 and 4 percent of the genome of some modern humans hails from Neanderthals, stocky hominids who lived between 130,000 and 30,000 years ago.
That 2010 study used DNA extracted from Neanderthal bones found in Croatia. With the new availability of a partial Neanderthal gene sequence from Croatia, Labuda and his team had something to compare their mysterious X chromosome fragment with.
Using DNA from 6,092 modern X chromosomes from every continent, the researchers found that the modern-day fragment matches one found in the Neanderthal genome. [Read: Who Were the Neanderthals?]
In the past there have been numerous theories for the cause(s) of autism, Asperger’s syndrome, ADHD and Tourette syndrome. Most of these theories can at best explain small parts of these diverse syndromes. Many of them extend their findings in spectacular ways to be able to claim to explain larger parts of the autism spectrum with little success.
This theory approaches the problem from a new radical viewpoint. Instead of approaching autism as a disorder, brain defect or the result of poor socialization or parenting, it claims that autistics are fully functional.
All the areas that are central to autism are related to species-typical adaptations that vary widely between species. These include nonverbal signals, social organization, sensory acuteness, motor skills, general preferences, sexuality, physical traits and biological adaptations. Some of this diversity in autistics is poorly understood and virtually unresearched and therefore is not published in peer-reviewed journals. Because of this lack of research, Aspie-quiz, an online questionnary, is heavily referenced for these traits.
Recent genetic research have demonstrated that the Out-of-Africa (OoA) model with no interbreeding fails to explain nuclear DNA diversity in Eurasia. Several models of interbreeding that do explain this diversity exists today. It therefore is quite likely that Neanderthals contributed to the Caucasian genome. Aspie-quiz have demonstrated in a large survey in the US population that Afroamericans have only 1/6 of the autism prevalence of Caucasians. The same survey also indicates that Asians and American Indians have about 1/2 of the autism prevalence of Caucasians.
Principal Component Analysis (PCA) of Aspie-quiz yields axises that seems to be related to the first Eurasian Homo, the formation of modern humans in Africa or South Asia and the hybridization between modern humans and Neanderthals in Europe. These axises seems to be 1.8 million years, 150,000 years and 37,000 years, which fits pretty good with the archaeologic evidences available.
THE REAL EVE – THE DISCOVERY CHANNEL (2002)
The Real Eve is the title of a popular science book written by Stephen Oppenheimer and a documentary based on the book. The book is largely based on the “Out of Africa theory” of human origins. Oppenheimer uses information from various disciplines including genetics, archeology, anthropology and linguistics to synthesize theories on the origin of modern humans and their subsequent dispersal around the world.
The Eve in the title refers to Mitochondrial Eve, a name used for the most recent common ancestor of all humans in the matrilineal (mother to daughter) line of descent.
In the book, Oppenheimer supports the theory that modern humans first emerged in Africa and that modern human behavior emerged in Africa prior to the Out of Africa migration. Oppenheimer writes that there was only one migration out of Africa that contributed to the peopling of the rest of the world. Oppenheimer believes that anatomically modern humans crossed the Red Sea from the Horn of Africa and followed the “southern coastal route” once in Asia. Thus Oppenheimer is opposed to the theory that there was another out of Africa migration using northern route along the Nile and into the Levant as suggested by Lahr and Foley 1994. The book also supports the theory that modern humans were in South Asia during the Toba Castrophe. Oppenheimer uses familiar names to describe genetic lineages. The biblical analogies of Adam and Eve are used to describe the Most recent common ancestors via mitochondrial DNA and the y-chromosome. Other male lineages are described as Cain, Abel and Seth. Mitochondrial DNA haplogroups are frequently described with female names from regions where the haplogroups are common. For example haplogroup M is named Manju as it is frequent in India, and Haplogroup N is named “Nasreen” as it is predominant in Arabia.
The documentary, also known as Where We Came From in the United Kingdom, was released in 2002. The documentary was produced by the American cable TV network the Discovery Channel and was narrated by Danny Glover and directed by Andrew Piddington.
Blood does not lie and history is mostly lies. In an attempt to reconstruct what I can prove after abandoning what has been deemed false or proven to be unproven, I am now ridding myself of the dead weight and am moving straight into the real history I have once been kept from having easy access to.
The Sumerian civilization emerged upon the flood plain of the lower reaches of the Tigris and Euphrates Rivers about 4000 B.C. The social structure of the Sumerians was decidedly different from other societies of that and later times. The Sumerian communities were city states organized around a temple and ruled by a priesthood. The bulk of the people of the community were considered to be the servant-slaves of the god of the temple. The insecurities of life justified the role of the priesthood. When calamities occured despite the best efforts of the priesthood this was explained as being the result of the actions of other gods acting in concert which over-ruled the wishes of the local god.
The Berbers live in the Northern part of Morocco. They have the highest percentage of rhesus negatives.
Berbers are the indigenous peoples of North Africa west of the Nile Valley. They are discontinuously distributed from the Atlantic to the Siwa oasis, in Egypt, and from the Mediterranean to the Niger River. Historically they spoke various Berber languages, which together form a branch of the Afro-Asiatic language family. Today Arabic is spoken almost universally by Berbers, along with Darija, as well as French (in Morocco, Tunisia and Algeria) and some Spanish (in Western Sahara and parts of Morocco), due to European colonisation of the Maghreb. Today most Berber-speaking people live in Morocco, Algeria, Libya, Mali and Niger.
Berbers represent the major ethnic origin in North Africa, although up to perhaps a certain extent interbred with other elements (Arab, Subsaharian, Iberian , Punic…), but only about half of the Moroccan population and a third of the Algerian can be identified nowadays as Berber by speaking a Berber language (see there for estimates). Nevertheless, the culture of many Arabic-speaking ethnic groups in these countries is very similar to that of their Berber neighbours and often language may be the only difference between Berbers and Arabs in the Maghreb. Thus, very high estimates of Berber population might include ethnic groups which no longer speak a Berber language. There are also smaller Berber populations in Libya and Tunisia, though exact statistics are unavailable  and very small groups in Egypt and Mauritania. Tuareg Berber spread southwards to Mali, Niger and Burkina Faso. Some 600,000 Tuareg Berbers live in Mali and 400,000 in Niger. Prominent Berber groups include the Kabyles of northern Algeria, who number about 4 million and have kept, to a large degree, their original language and culture; and the Shilha or Chleuh (French, from Arabic Shalh and Shilha aš?l?i) of south Morocco, numbering about 8 million. Other groups include the Riffians of north Morocco, the Shawiya language of Algeria, and the Tuareg of the Sahara. There are about 2.2 million Berber immigrants in Europe, especially the Riffians and the Kabyles in the Netherlands, Belgium and France.
Only around 800 kilometers from the Atlas mountains where the Berbers originate is where you can find the Basque region.
35 000 years ago the men of southern France and the Basque region hunted the wild bison, wooly rhinocerus, horse, and mammoth where they lived in tepees with the women and not in the painted caves according to popular belief. The women would have gathered wild fruits, seeds, and berries where they brought them back to thier campsite. They probably spent most of thier time in those dark tepees and only occasionaly did they most likely wander out of thier tepees to collect the fruit of the plains. The reason being for this is that after menstruation and child birth, they needed protection from the cold and other weather elements to raise and feed thier children. This is probably where the women, over thousands of years, obtained the recessive genotypes like lighter skin and hazel eyes, although not necessarily the dominate phenotype of the O+ blood factor which they most likely picked up 5-6000 years earlier when they ventured out of Siberia on thier way to northern and southern Europe. It is positively sure that a few of the O rhesus positive women joined the O rhesus negative tribe, but a lot of the women travelling to southern Europe just below the Swiss alps likely still had the O rhesus negative factor while those travelling north of the glaciated Swiss alps likely had the O rhesus positive factor. This may explain why a lot of Spanish and Italians presently have dark hair as opposed to the Germans and French who have lighter complexions and blond or blonde hair.
The Caucasus region from which the term “Caucasian” originates.
And now the Celts:
This map shows
yellow: the core Hallstatt territory, expansion before 500 BC
light green: maximum Celtic expansion by the 270s BC
very light green: Lusitanian area of Iberia, “Celticity” uncertain
intermediate green: the boundaries of the six commonly-recognized ‘Celtic nations’, which remained Celtic speaking throughout the Middle Ages (viz.Brittany, Wales, Cornwall, Isle of Man, Ireland, Scotland)
dark green: areas that remain Celtic-speaking today
The Celts (pronounced /?k?lts/ or /?s?lts/, see pronunciation of Celtic) were a diverse group of tribal societies in Iron Age and Roman-era Europe who spoke Celtic languages.
The earliest archaeological culture commonly accepted as Celtic, or rather Proto-Celtic, was the central European Hallstatt culture (ca. 800-450 BC), named for the rich grave finds in Hallstatt, Austria. By the later La Tène period (ca. 450 BC up to the Roman conquest), this Celtic culture had expanded over a wide range of regions, whether by diffusion or migration: to the British Isles (Insular Celts), the Iberian Peninsula (Celtiberians, Celtici and Gallaeci), much of Central Europe, (Gauls) and following the Gallic invasion of the Balkans in 279 BC as far east as central Anatolia (Galatians).
The earliest directly attested examples of a Celtic language are the Lepontic inscriptions, beginning from the 6th century BC. Continental Celtic languages are attested only in inscriptions and place-names. Insular Celtic is attested from about the 4th century AD in ogham inscriptions, although it is clearly much earlier. Literary tradition begins with Old Irish from about the 8th century. Coherent texts of Early Irish literature, such as the Táin Bó Cúailnge (The Cattle Raid of Cooley), survive in 12th-century recensions. According to the theory of John T. Koch and others, the Tartessian language may have been the earliest directly attested Celtic language with the Tartessian written script used in the inscriptions based on a version of a Phoenician script in use around 825 BC.
By mid 1st millennium AD, following the expansion of the Roman Empire and the Great Migrations (Migration Period) of Germanic peoples, Celtic culture and Insular Celtic had become restricted to Ireland and to the western and northern parts of Great Britain (Wales, Scotland, Cornwall and the Isle of Man) and northern France (Brittany). The Continental Celtic languages ceased to be widely used by the 6th century.
Insular Celtic culture diversified into that of the Gaels (Irish, Scottish and Manx), the Brythonic Celts (Welsh, Cornish, and Bretons) of the medieval and modern periods. A modern “Celtic identity” was constructed in the context of the Romanticist Celtic Revival in Great Britain (Wales, Scotland, Cornwall and the Isle of Man) and Ireland. In France a similar revival of Breton is taking place in Brittany.
Ireland and Scotland have large amounts of rhesus negatives and so do the Welsh. So how about the Brits? They are Germans. Saxons.
Many have alleged the Pharaos being descendants of the Irish or Scots and a high rhesus negative ratio being an indicator thereof. Also a lot of parallels between Atlantis and the British Isles have been established.
Are there a few original human tribes which are rhesus negative and their ruling over the earth has been maintained for all this time?
Disclaimer: The below list of Rhesus Negative Celebrities is for entertainment purposes only and the Rhesus Negative status of all celebrities cannot be guaranteed 100 percent other than on me relying on the data I am provided with which always checks out as credible.
This list has been composed in good faith and each listing is based on references from sources which deem credible.
If you believe that any of the listed celebrities doesn’t belong on the list, please state your case below and provide a link if possible.
If any of you has another celebrity or number of celebrities which are rhesus negative to the best of your knowledge, please comment on this album and provide a link as well.
Thank you for joining Rhesus Negative Bloodtype!
B+ about B-ing –
Michael Dammann and the crew
If you know of a Rhesus Negative Celebrity I have not yet added, you can comment below or see me on Facebook:
Getting information as to which celebrities are rhesus negative obviously is not easy. But many of them are stating it publicly, others tell some of us in private and even more are being overheard talking about it.
Ellenor Whitty and Dan Aykroyd. Ellenor is 0- and Dan is AB-
Autopsy reports often limit the blood type to the letter(s) without the – or + and even looking at Jimi Hendrix’ military “dog tags” you only see the 0.
So anybody with information in regards to which celebrities are rhesus negative, please forward me the information.
Here is my compilation in regards to those who are or have been in the public eye and are to the best of my knowledge rhesus negatives:
Prince Charles 0 -
Queen Elizabeth II 0-
Former President Eisenhower Type O-Neg
Former President John F. Kennedy Type AB-Neg
Former President Richard Nixon Type O-Neg
Former President Ronald Wilson Reagan
Former President George W. Bush Sr. Type A-Neg
Former President Bill Clinton AB-Neg
Current President Barack Hussein Obama AB Negative
Barry Goldwater A-
Actors and Actresses:
Dan Aykroyd Type AB-Neg
Johnny Depp B Negative
Leonardo Di Caprio B-
Mia Farrow B-
Marilyn Monroe was Type AB-Neg
Fox Mulder “X-files” Type O-Neg
Paul Newman 0-Neg
Jack Nicholson B-
Jimi Hendrix 0-
Mick Jagger AB-
John Lennon 0-
Paul McCartney B- (Identify questioned)
Freddie Mercury R.I.P. (Farrokh Bulsara)
Elvis Aaron Presley 0 negative
Ringo Starr A negative
Peter Steele (Band “Type 0 Negative”) 0-
Sting (Gordon Summer) 0-
Pharaoh Ramses II Type B-Neg (He also was HLA-B27 positive, according to his mummy. They did all kinds of DNA testing on him to confirm)
Milton William Cooper (May 6th, 1943 – Nov 5th, 2001)
Sir Arthur Conan Doyle (Sherlock Holmes)
Robert A. Heinlein
Zacharia Sitchin Type Neg
Brad Steiger O-Neg
Erik Von Daniken Type O-Neg
Robert Anton Wilson Type Neg
Sir Isaac Newton
Charles de Gaulle (Former President of France)
Senator John McCain O-Neg
Shroud Of Turin was AB-Neg
Dr. Martin Luther King
Malcolm X (Malcolm Little)
O.J. Simpson is Type A-Neg
High profile murder victims:
Ron Goldman Type O-Neg
Laci Peterson Type O-Neg
Vlad, the Impaler (Dracula)
Adolf Hitler AB –
Other people in the public eye:
John F. Kennedy Jr.
David Rockefeller, Sr.
Much of the above research has been conducted with the help of the member of the following communities:
“[Libby] was born 6½ weeks early with Rh Disease. It happens when the mother is Rh-negative and the baby’s Rh-positive. It’s a very serious thing, and we were in and out of specialists’ offices every other day. Libby came out needing a partial [blood] transfusion and was in an incubator for 2½ weeks — we couldn’t touch her, nothing. That first month was brutal.”
As more and more studies are being focused on the rh negative blood factor, more and more differences in regard to health related subjects come to light.
The latest one:
RhD phenotype modulates the influence not only of latent toxoplasmosis, but also of at least two other potentially detrimental factors, age and smoking, on human behavior and physiology. The negative effect of smoking on health (estimated on the basis of the self-rated number of common viral and bacterial diseases in the past year) was much stronger in RhD-negative than RhD-positive subjects. It is critically needed to confirm the differences in health response to smoking between RhD-positive and RhD-negative subjects by objective medical examination in future studies.
Source: Flegr J, Geryk J, Volný J, Klose J, ?ernochová D (2012) Rhesus Factor Modulation of Effects of Smoking and Age on Psychomotor Performance, Intelligence, Personality Profile, and Health in Czech Soldiers.
Your eyes are tiny spheres of wonder. A doctor can find warning signs of high blood pressure, diabetes, and a whole range of other systemic health issues, just by examining your eyes. Ophthalmologist Neal Adams explains why the eye’s tissues and blood vessels make such a good barometer for wellness.
In order to calculate how many rh positives in a country are rh negative recessively and therefore able to pass it on to have rh negative offspring, scientists worldwide are using the Hardy-Weinberg principle to determine the number of rh negative homozygotes, rh positive homozygotes (Rh +/+ individuals) as well as rh positive heterozygotes (Rh +/- individuals).
Let´s use the table of rh negative gene frequencies below as an example:
I have underlined the rh negative gene frequency of the Spanish Basque population. 0.532 is the rh negative gene frequency marked as r(cde) which is 53.2%.
Let´s see the formula:
x=0.532, so 0.532² gives us the Rh Negative Frequency of 0.283024 meaning the percentage of rh negatives in the Spanish Basque population is 28.3%.
The number of rh positives not carrying the rh negative gene recessively is determined by (1-x)² so (1-0.532)² is 0.219024 meaning 21.9% of Spanish Basques are rh positive homozygotes.
And 2x(1-x) being 2×0.532x(1-0.532) means that 0.497952 or around 49.8% of Spanish Basques are rh positive, but carry the rh negative gene recessively.
A lot of recent research highlights many of our suspicions being in fact more that just amateur observations. At this point and time we have began a brandnew journey where blood factor and medical problems are coming to the forefront in terms of a connection no longer being deniable.
If you would be interested and ready to participate in an official study with a leading researcher in the field, please contact us here:
I need your email address, name, blood type (both rh factor and ABO).
If you are selected to become part of the study, you will be sent a questionnaire.
Thank you in advance. Here is the highlight from a previous study:
About one in ten women who have a child with autism have immune molecules in their bloodstream that react with proteins in the brain, according to a study published 20 August in Molecular Psychiatry1.
Several research groups have found these immune molecules, called antibodies, in mothers of children with autism, and have shown that prenatal exposure to the antibodies alters social behavior in mice and monkeys.
The new study, which includes more than 2,700 mothers of children with autism, is the largest survey yet on the prevalence of these anti-brain antibodies.
“It’s a very large sample size,” says study leader Betty Diamond, head of the Center for Autoimmune and Musculoskeletal Disorders at The Feinstein Institute for Medical Research in Long Island, New York. The scale gives a clearer impression of the prevalence of these antibodies, she says.
Antibodies help the body’s immune system recognize and fight off disease-causing microorganisms such as bacteria and viruses, but sometimes the body mistakenly produces antibodies to its own proteins. In some people, this results in autoimmune diseases such as rheumatoid arthritis and lupus, in which the body attacks its own tissues.
Researchers say anti-brain antibodies do not harm the brains of the women who produce them because of the blood-brain barrier, a filter that prevents most molecules from entering the brain. But the immature blood-brain barrier of a developing fetus may let them through, allowing them to damage the brain and perhaps cause autism.
Diamond’s team also found that women who have autism-linked antibodies are more likely to have other markers of autoimmunity compared with those who don’t carry these antibodies. Studies have shown that women with an autoimmune disease also have an increased risk of having a child with autism2.
“This ties together the epidemiological finding that women who have autoimmune disease are more likely to have kids with autism, with the idea that there are actually antibodies against fetal brain in their serum,” says Paul Patterson, professor of biology at the California Institute of Technology, who was not involved in the work.
In the new study, Diamond’s team screened blood plasma samples from 2,431 mothers enrolled in the Simons Simplex Collection (SSC). The SSC is a registry of families with one child affected by autism and unaffected parents and siblings, and is funded by SFARI.org’s parent organization.
The researchers found that plasma from 260 of the women, or 10.5 percent, reacts strongly with mouse brain tissue, a signal that the blood contains anti-brain antibodies.
They also screened samples from 318 mothers enrolled in a different autism registry, the Autism Genetic Resource Exchange, and found that 28, or 8.8 percent, of them also have anti-brain antibodies.
In contrast, among a group of 653 controls drawn from the general population of women of childbearing age in New York City, only 17, or 2.6 percent, carry the autism-linked antibodies.
This means that the prevalence of anti-brain antibodies is about four times greater among mothers of children with autism than among the controls.
However, “The control population could have had mothers of children with autism in it,” notes Judy Van de Water, professor of clinical immunology at the University of California, Davis, who was not involved in the work. In fact, some of the women in the control group may not have children at all.
Still, Van de Water says, the study “corroborates, from an independent investigator, that women in this population have a higher incidence” of the antibodies.
Van de Water has found anti-brain antibodies in autism mothers who are part of the Childhood Autism Risk from Genetics and the Environment study using a different methodology3, known as Western blotting, and says that the differences in study design make the similar results more persuasive. “It’s further support for this potential pathway to autism,” she says.
However, she cautions that measuring reactivity to mouse brain tissue also has drawbacks, because of the action of chemicals used in preparing the brain tissue for analysis. “The perfusion and fixation process that you have to go through is going to alter some of the antigens,” the proteins to which the antibodies bind, she says. This may ‘mask’ some of the binding patterns that occur in live brains.
Using the mouse brain enabled Diamond’s team to pinpoint which regions of the brain the antibodies affect. They found that the antibodies bind primarily to neurons in the frontal cortex, hippocampus and cerebellum, all areas that have been implicated in autism. Fetal findings:
Patterson notes that the researchers tested binding of the antibodies to adult mouse brain, but protein expression in the adult brain is often very different from that in the fetal brain. “Optimally, one would like to know what the binding is to fetal human brain or fetal monkey brain,” he says.
According to Diamond, the team did find that the antibodies also bind to fetal human brain extracts and to whole fetal mouse brain. However, she says, it wasn’t possible to see which regions of fetal mouse brain they bind to because the brains are too small.
Her team performed several other analyses that uncovered tantalizing links between autism-linked antibodies and autoimmunity more generally.
First, they screened the blood of study participants for anti-nuclear antibodies (ANAs), which bind to molecules found in the cell nucleus and are commonly found in people with a variety of different autoimmune disorders. They found that 52 percent of autism mothers who carry anti-brain antibodies also have ANAs. In contrast, only 13.4 percent of autism mothers without anti-brain antibodies have ANAs, as do 15 percent of controls.
Mothers with anti-brain antibodies are also more likely to have autoimmune diseases, especially rheumatoid arthritis and lupus, compared with mothers who don’t have these antibodies.
Finally, the team screened a group of 363 women with rheumatoid arthritis for autism-linked antibodies. They found that 13.5 percent of these women have anti-brain antibodies, similar to the prevalence among mothers of children with autism and much higher than that of controls.
That’s a rather puzzling finding, Patterson says. “If they’re just as likely to be found in rheumatoid arthritis women, that means they’re not especially specific to autism,” he says.
But a link between rheumatoid arthritis and autism isn’t unheard of, Diamond says. In a separate series of experiments, she has found that antibodies that cause kidney damage in women with lupus also cause cognitive impairment in mice exposed to the antibodies in utero4.
Diamond says she has unpublished data indicating that some of the brain proteins that bind the antibodies are already implicated in autism or other neurodevelopmental disorders — but declined to reveal details. Are any of the targets she has identified the same as those reported by Van de Water and her colleagues in July? “It’s sort of interesting,” Diamond says. “They’re not.”
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What is the blood type of the child?
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Toxoplasma gondii, a parasite infecting 20-60% of humans in various countries, influences the behaviour of infected animal and human hosts. Infected human subjects have changes in several of Cattell’s and Cloninger’s personality factors. Recently, three independent studies have shown that Rh-positive subjects are protected against the T. gondii-induced changes of reaction times and increased risk of traffic accidents. Here we searched for evidence of similar effects of RhD phenotype on toxoplasmosis- or aging-associated changes in the personality profile of about 302 blood donors. We found that Rh-positive and Rh-negative subjects responded differently to toxoplasmosis. In addition to the already known effects of toxoplasmosis on novelty seeking, self transcendence, superego strength and protension, we also found effects of RhD phenotype on ego strength, protension, and praxernia, as well as opposite effects of toxoplasmosis on ego strength, praxernia, ergic tension and cooperativeness in Rh-positive and Rh-negative subjects. Moreover, our results indicate that RhD phenotype might influence not only the effect of toxoplasmosis but also the effect of aging on specific personality traits.
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